The so-called special type of the disease that is not typical clinical manifestations of the disease a total of five following : (1) atypical measles syndrome patients were vaccinated against measles after a number of years blood antibody levels decline reinfection natural measles virus and onset. Was particularly prevalent in the 10-24-year-old youth winter and spring distributed free of infectious. Include sudden high fever headache abdominal pain and muscle pain but no cough runny nose or conjunctivitis phenomenon. Incidence 48 ~ 72 hours rash common in the ankle wrist and physically fold Department. Skin rash over point for the blockage a small number of spotted expanding rash or blistering wind Mission in 3 ~ 5 days and gradually spread to the head from the trunk and limbs. Volar Ministry may have edema the palate can blockage but no Coriolis spot. Vaccinations past rashes more obvious. Skin rash often dissipated within two weeks. Complications of pneumonia pleural effusion hilar lymphadenopathy encephalitis persistent joint pain myocarditis such as geriatrics. (2) Heavy measles is more common in serious secondary infection nutrition or extremely bad and those suffering from other ailments. Systemic symptoms of heavy high fever of 40 ° C to 41 ° C convulsions coma very dangerous. Rashes or dense integration deep color; Scarce or missing color bleak. This type of poor prognosis prone to cause death. (3) light measles fever and a rash spacing ~ 7 days. A rash two days ~ generations. Measles from scratch the surface diffusion in the trunk limbs rarely pink rash the rash is regressive On pigmentation. Grouper oral mucosa were not large only see oral mucosa rough congestive. (4) Adult Children of measles and measles rash similar but the Coriolis spot rare. Systemic symptoms are serious but less complications. (5) Herpes-type measles clarify the blister fluid can become bullosa. A very small number of cases of haemorrhagic herpes it may erythema multiforme or toxic epidermal necrolysis.
Acute mastoiditis gas is the mastoid bone and the mucosa of acute suppurative inflammation by the more acute otitis media development purposes. Children styles. Its etiology and pathology : as body resistance weak strong assortment of drugs improper handling or treatment otitis media with the continued development and drums imported sinus swelling of the mucous membrane was blocked the mastoid impeded drainage of abscess mastoid gas accumulation in the room filling the mucosal necrosis bone wall due to oppression and self Pott inflammatory lesions occurred decalcified necrosis and ulceration of the separated the gas integration forming a large vomica said integration mastoiditis or mastoid pyometra. If hemolytic streptococcus or influenzae induced acute mastoiditis mastoid bone multi-wall intact filling the room with bloody discharge bleeding mastoiditis said. Although acute otitis media were treated but because the amount of antibiotics or to complete treatment although mastoiditis lesions continue to develop and the general and local symptoms is not clear that it would not be discovered hidden called mastoiditis. Acute mastoiditis if not brought under control continued development of inflammation may break the mastoid bone wall meningitis complications.
Otogenic complications in the diagnosis pay attention to the following two grounds : (a) in the diagnosis of otogenic : ?٠from history have ears discharging history. Especially chronic suppurative otitis media cholesteatoma purulent secretions are covered shaped like scums; Bone ulcers otitis media over its Pott with bloodshot. In complications before discharging more sudden decrease or stop or sudden increase in the former prompt drainage blocked. The latter said to more acute; and many ear pain general malaise fever headache anorexia and so on. ?ڠfrom ear examination many of the relaxation of the tympanic membrane or marginal perforation may also tense as large perforation; Visibility indoor drum granulation or polyps; Drum sinus or mastoid skin redness tenderness. ?۠who has been suffering from ear extracranial complications arise such as the ears after subperiosteal abscesses deep neck abscess intracranial there are also cases such as high fever severe headache chills muscle twitching nausea vomiting neck stiffness often suggested as Otogenic. ?ܠmastoid film or temporal bone CT scanning multi-bone destruction as sigmoid sinus bone plates or drum sinus days cover the mastoid bone destruction. (2) the diagnosis of complications : According to various complications of clinical characteristics with the necessary special examinations open to the general diagnosis. But several complications arise simultaneously or successively the symptoms and signs complex and therefore in a specific phase of the disease it is difficult to determine whether one of the main complications. In addition the blind abuse of antibiotics diseases of the masking effect so that clinical manifestations become typical lost its inherent laws and will increase diagnosis difficult. Therefore attention should be paid to closely observe the patient's condition according to the need for fundus examination. Brain CT scan and MRI (magnetic resonance) and other special examinations characterization of disease diagnosis is of great value.
Otogenic complications of surgery is the main treatment. The purpose of surgery is to remove lesions and open drainage. From the mastoid exploration mastoidectomy and others (including exposure dural sinus wall and the side of the widening mastoid surgery) to intracranial abscess drainage and removal its depends on the type and extent of the complications of the type and characteristics. Surgical treatment in the meantime there can be no underestimating the role of antibiotics. It should be noted in a timely adequate and in the light of the results of bacteriological choose ampicillin erythromycin cefazolin penicillin ciprofloxacin (normally not for children) chloramphenicol and so on. Intracranial complications should adopt the above two kinds of combination of antibiotics intravenous infusion in the main. Otogenic complications particularly intracranial complications are generally more unconscious fever chills headache nausea and even vomiting application : (1) support therapy infusion blood transfusions or plasma and added composite amino acids such as albumin. (2) the appropriate use of steroids such as dexamethasone 10 ~ 20 mg / day intravenous drip. (3) intracranial hypertension dehydration reducing the intracranial pressure is also very important. If each with a 20% mannitol Co. ~ 50% and ~ 40 ml glucose. Quick turn intravenous infusion every 4 ~ 6 hours a time records access volume and attention should be given to water and electrolyte balance.
- Dan scored herpetic keratitis it should prompt medical treatment. Use drugs surgical methods. Drug treatment main selection of antiviral drugs such as ribavirin also known as ribavirin for the broad-spectrum antiviral efficacy than the law and the normal cell toxicity is low acyclovir also known as acyclovir to a more effective antiviral drugs especially for herpes simplex virus a marked inhibition; Other antiviral drugs such as adenosine A sugar trifluoromethyl - thymidine other. May choose adrenocorticotropic hormone as it inhibit corneal immune response and anti-inflammatory effect commonly used in the herpes simplex virus keratitis late treatment but should grasp the following principles : a superficial corneal epithelial inflammation or banned. Because of the virus and can be activated collagenase activity and the promotion of virus propagation the development of lesions to the deep. It can also inhibit epithelial regeneration and even ulcer perforation. 2 only for deep ulcers inflammatory response without those. HSK especially deep because discoid keratitis is the pathogenesis of both viral antigens allergic factors. Therefore application of glucocorticoid is reasonable but it should be applied to antiviral drugs. 1-2 daily were stained with fluorescein and when ulcers appeared and immediately suspended by ulcer treatment.
Bubbles conjunctivitis is an allergic inflammatory reaction the majority are female incidence of children and young people particularly the partial eclipse malnutrition physical infirmity or with a history of tuberculosis often associated with facial eczema and lymph nodes it is also called eczema or scrofula M colon keratitis is caused by microbial delayed hypersensitivity and the body of Mycobacterium tuberculosis. Staphylococcus. Koch-Weeks of Bordetella bronchiseptica and other microbial infection. It was also suggested that intestinal parasites also can cause allergic reactions such inflammation. The most common disease in the limbus or translucent hygiene miliary large round gray and white or light yellow small round bubbly uplift a diameter of about 2-3mm. Congestive bubble around limitations squeezing hands when no sense of pain and a few days later nodules epithelial exfoliation the top forming small ulcers generally self-healing. Initial patients may develop mild eye discomfort when invasive corneal disease can occur photophobia of eyes tears and eye pain and other symptoms. Its occurrence in the conjunctiva were no scar left who was born in the Department of translucent healed scar left Born in the corneal ulcer margin to the progressive development of central corneal opacity into bundles conjunctival blood vessels are entering the beam to the beam-ulcer road called bundles keratitis. Shallow scleritis and more generally with rheumatoid arthritis nodules or erythema with other connective tissue diseases many of bilateral. Acute disease the development has been slow but easy to relapse the patient consciously photophobia of eyes tears pain but there are not obvious general vision may not be affected. Ocular conjunctiva of the ball or marginalized parts of the cornea can be seen congestive lentils size of the uplift the limitations were purple nodules sclera surface is vasodilator performance. Nodules tenderness which is due to ciliary nerve stimulation induced. The rapid development of nodules per persistent inflammation a few weeks after the change-nodules or color to white and finally completely dissipated inflammation. Mild Depression left the surface and a dark gray color and bulbar conjunctival adhesion at the scene. Bubbles conjunctivitis prognosis is better after about 10 ~ 14 days can be self-healing the treatment of partial use of corticosteroid. If 0. 1% dexamethasone eye drops administered topically but also in the bulbar conjunctival injection. In addition we must strengthen nutrition physical activity to improve their physique taking cod liver oil capsules vitamin B2 calcium and so on. If more stubborn viable nodular partial freeze. Xiefei Chinese medicine treatment for heat-Sanjie.
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